![]() ![]() We cannot simply duplicate with detailing, because we need this area to have a new associated level.Īs the original question asks: Why does Revit no longer allow us to copy tags from one view and into another. In this situation it would be great to copy tags from view to view, but as the original question mentioned.this does not work. Due to the complexities of view ranges and associated levels, there is a separate level that covers this vast region of the concourse. picture this: You're walking along the concourse and you are at elevation 385', you walk down a ramp and you are STILL on the "Concourse level" but you are now at elevation 375'. We have a complicated building (Port of Seattle) with multiple different elevations of a single level due to ramps up and down. This one is actually a situation in which "Duplicate with Detailing" does not work. So here's my question in regards to an almost identical situation. The only differences are phase and phase filter. Both views have the same crop, the same annotation crop, the same view range, the same filters, worksets, etc. Is there something different with Revit, or something that could have changed with my company's template?Īnd just to head these things off: Yes, the elements are visible in both views. By 'Lately' btw, I mean since using MEP 2015. I just get the error, but nothing actually copies. However, the rest of the stuff would copy. "Some tags were not copied because they could not find a host." which makes sense, because some of the stuff was demolished and doesn't appear in the new work view. I used to be able to do a big window selection in the demo view, filter the selection to just the tags then just Ctrl+C, and past-aligned-to-same-place in the new and they'd show up. It can take a long time to tag all the ducts, pipes, air terminals, equipment, etc. and the only difference between the two is the phase. I have two views, with identical ranges, extents, etc. However, the Tn 6552 tnsD targeting gene was interrupted by an ISAba12, and Tn 6552 is not downstream of glmS.This is something I used to be able to do very easily, but hasn't been working lately (Revit 2016). A related transposon, designated Tn 6552, was detected in ATCC 17978 (ST437) and other ST437 isolates. The GC1 isolates were mainly lineage 2, but a potential third lineage was also detected. A recombination-free phylogeny revealed that there were several transposon acquisition events in GC1. baumannii lineage, indicating that the transposon may have been acquired by replacement of a segment of the chromosome. However, in some of these isolates the surrounding glmS region was clearly derived from a different A. baumannii isolates belonging to GC1, GC2 (ST2) and ST10. Tn 6171 or minor variants were detected in the equivalent location in complete or draft genomes of several further A. Tn 6171 is bounded by 29 bp inverted repeats and, like Tn 7, includes additional TnsB binding sites at each end. baumannii global clone 1 (GC1) lineage 2 isolate D36, includes tns genes encoding proteins related to the TnsA, TnsB, TnsC transposition proteins (50–59 % identity), TnsD targeting protein (43 % identity) and TnsE (31 % identity) of Tn 7, and is found in the chromosome downstream of the glmS gene, the preferred location for Tn 7, flanked by a 5 bp target site duplication. ![]() ![]() Here, large novel transposons that carry genes for synthesis and transport of the fimsbactin siderophores present in some A. Acinetobacter baumannii is a successful opportunistic pathogen that can compete for iron under iron-limiting conditions. ![]()
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